This study was designed to assess the extent to which psychological stress influences the experience of asthma-like symptoms (e.g., shortness of breath, coughing) and lung function (e.g., daily peak flow variability) in the workplace, and if so, by what mechanisms.
Reach a greater understanding of the potential moderating effects of stress on the development of occupational asthma, and to greater recognition of how psychological stress in the workplace is an important risk factor in this population.
§ Full scientific description :
The goal was to investigate the extent to which daily psychological stress influences the experience of asthma-like symptoms (e.g., wheezing) and lung function (daily peak expiratory flow variability), and the mechanistic role the sympathetic nervous system (SNS) plays in these relationships. It was hypothesized that an increase in negative emotions would precede the experience of asthma-like symptoms and a reduction in lung function. It was also hypothesized that this relationship would be mediated by alterations in parasympathetic and sympathetic control.
§ Timeline: 2009-2011
§ Project Personnel
Main Investigator: Simon L. Bacon, Ph.D.
§ Primary Contact Person
Maxine Boudreau, Ph.D.(c), Project Coordinator
Asthma in the Workplace Centre, Canadian Institutes of Health Research (CIHR)
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